Iga nephropathy occurs due to external invasion of inflammatory substances
cause kidney damage, which in the course of treatment would be
anti-inflammatory, inhibiting inflammatory substances continue to kidney
inherent cell is mesangial cell damage. External inflammatory substances into
the human body and iga generate antigen-antibody immune complexes, and then
deposited in the mesangial area of renal ischemia and hypoxia occurs, and thus
to do is be vasodilators improve blood circulation and kidneys ischemic hypoxia
for repair mesangial cells provide a good therapeutic environment. So iga
nephropathy four Yan is not serious it?
IgA nephropathy Lee grade Ⅳ grade: severe glomerular lesions were diffuse
mesangial proliferation and sclerosis, some or all of glomerulosclerosis,
visible crescent (<45%). Tubular atrophy, interstitial infiltration, and
occasionally renal interstitial foam cells. Typing this crescent take an
example: this type crescent close to 50%, can be said that if patients do not
pay attention, it will most likely achieve this figure, but studies have shown
that when the crescent exceeds glomerular When more than 50% of the total, is
the crescent glomerulonephritis (or crescent iga nephropathy), which is other
than domestic lupus nephritis most common type II - immune complex
glomerulonephritis crescent.
From the above description, it can be seen iga nephropathy in four stages,
are more serious, and therefore need to be treated as soon as possible
specification. Increase blood viscosity will increase in inflammatory cells,
then platelets hyperthyroidism, enhanced coagulation, leading to microthrombi
constituted, in order to prevent micro thrombus composition, and thus to
anticoagulant therapy, keeping blood unobstructed, avoid immunity composite
excessive deposition.
Through the above description everyone iga nephropathy four Yan is not
serious all clear now! If you have any questions about the above, you can always
consult us by email: kidneyhospitalabroad@hotmail.com.
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